How to know if you were bitten by a tick with encephalitis and what are the symptoms?

How to know if you were bitten by a tick with encephalitis and what are the symptoms? - briefly

Check the bite site for a red, expanding rash and watch for fever, severe headache, neck stiffness, confusion, or seizures that develop within weeks of exposure. These neurological signs suggest a tick‑borne encephalitis infection.

How to know if you were bitten by a tick with encephalitis and what are the symptoms? - in detail

A tick bite that transmits encephalitic viruses can be identified through a combination of visual inspection, exposure history, and clinical signs. After removing the arthropod, examine the bite site for a small, dark, raised lesion (often called a tick bite scar) that may persist for several days. The presence of a red or inflamed area around the attachment point, especially if it expands or becomes painful, warrants further attention.

Key steps for assessment:

  • Review recent outdoor activities in regions where tick‑borne encephalitis (TBE) is endemic (e.g., forested or grassy areas of Europe and Asia).
  • Determine the time elapsed since the bite; the incubation period for TBE typically ranges from 4 to 28 days, with most cases appearing within 7–14 days.
  • Note any immediate reactions such as a local rash, swelling, or flu‑like discomfort occurring within 24–48 hours.

If encephalitic infection develops, a distinct set of systemic and neurological symptoms emerges. Early manifestations (often termed the “first phase”) resemble a nonspecific viral illness:

  • Fever ≥ 38 °C
  • Headache, often frontal or occipital
  • Muscle aches and fatigue
  • Nausea or mild gastrointestinal upset

These signs may last 3–7 days before a brief remission. The “second phase” involves central nervous system involvement and includes:

  • High fever persisting beyond the initial phase
  • Severe headache with photophobia
  • Neck stiffness indicating meningeal irritation
  • Confusion, disorientation, or reduced consciousness
  • Motor weakness, tremor, or ataxia affecting coordination
  • Cranial nerve deficits such as facial palsy or double vision
  • Seizures, particularly in severe cases

Laboratory evaluation supports the clinical picture. Serum or cerebrospinal fluid analysis typically shows:

  • Elevated white‑blood‑cell count with lymphocytic predominance
  • Increased protein concentration in CSF
  • Presence of TBE‑specific IgM antibodies; seroconversion to IgG confirms recent infection

Imaging (MRI) may reveal hyperintense lesions in the basal ganglia, thalamus, or brainstem, correlating with neurological deficits.

Prompt medical evaluation is essential when any combination of the above symptoms appears after a tick exposure, especially if the geographic risk is high. Early antiviral or supportive therapy, alongside monitoring for complications such as respiratory failure or long‑term cognitive impairment, improves outcomes.