How does the Barss spray work on fleas? - briefly
The spray delivers a neurotoxic insecticide (such as pyrethrin or fipronil) that penetrates the flea’s exoskeleton, disrupts nerve signal transmission, and induces rapid paralysis and death. Direct application to pets or environments provides immediate contact killing and residual protection for several weeks.
How does the Barss spray work on fleas? - in detail
Barss spray targets adult fleas and developing larvae through a combination of rapid‑acting neurotoxins and growth‑inhibiting compounds. The formulation contains a synthetic pyrethroid that binds to voltage‑gated sodium channels in the flea nervous system, forcing prolonged depolarization and resulting in paralysis within seconds. Simultaneously, an insect growth regulator (IGR) interferes with chitin synthesis, preventing eggs from hatching and larvae from molting.
Application delivers a fine aerosol that penetrates fur, bedding, and cracks in flooring. Droplets spread by capillary action, coating the host’s coat and surrounding environment. Upon contact, the pyrethroid penetrates the exoskeleton, reaching the central nervous system. The IGR, being less volatile, remains on surfaces for weeks, disrupting the life cycle after the initial kill.
Key operational steps:
- Deposition: Pressurized nozzle releases micro‑particles (10–30 µm) that settle uniformly.
- Absorption: Lipophilic agents dissolve in sebum, maintaining contact with fleas feeding on the host.
- Neurotoxic action: Sodium‑channel activation persists, causing loss of motor control and death.
- Growth inhibition: Chitin‑blocking molecules impede exoskeleton formation in eggs and larvae, halting population renewal.
Safety profile relies on selective toxicity. Mammalian sodium channels exhibit lower affinity for the pyrethroid, reducing systemic risk when used as directed. The IGR is classified as low‑toxicity for mammals and birds, but exposure to aquatic organisms should be avoided.
Resistance management recommends rotating Barss spray with products containing alternative modes of action, such as neonicotinoids or spinosad, to prevent selection of tolerant flea strains. Regular re‑application according to label intervals sustains efficacy, typically every 30 days for adult control and every 90 days for IGR persistence.